已结题科研项目
1、国家自然科学基金青年基金项目,81201472,转录因子E2F1在内皮祖细胞介导的创伤愈合中的作用及机制, 2013/01-2015/12,23万元,已结题,主持。
2、国家自然科学基金面上项目,81370581,B细胞活化转录因子(Batf)调节Th17在 肝移植排斥反应中的作用机制研究, 2014/01-2016/12,70万,已结题,参与。
3、国家自然科学基金面上项目,30972796,干扰素调节因子-4结合蛋白(IBP)调节 Th17细胞参与排斥反应的机制研究, 2009/01-2011/12, 30万,已结题,参与。
4、美国National Institutes of Health (NIH) R01课题, 1R01HL093439,E2F1 and Ischemic Neovascularization, 2009/04-2015/03,150万美元,已结题,参与。
代表学术论文
(1) Cheng M, Zhou J, Wu M, Boriboun C, Thorne T, Liu T, Xiang Z, Zeng Q, Tanaka T, Tang YL, Kishore R, Tomasson MH, Miller RJ, Losordo DW, Qin G*. CXCR4-mediated bone marrow progenitor cell maintenance and mobilization are modulated by c-kit activity. Circ Res. 2010 Oct 29; 107(9): 1083-93. (IF: 11.019)
(2) Wu Min*, Zhou Junlan, Cheng Min, Boriboun Chan, Biyashev Dauren, Wang Hong, Mackie Alexander, Thorne Tina, Chou Jonathan, Wu Yiping, Chen Zhishui, Liu Qinghua, Yan Hongbin, Yang Ya, Jie Chunfa, Tang Yao-Liang, Zhao Ting C., Taylor Robert N., Kishore Raj, Losordo Douglas W., Qin Gangjian, E2F1 suppresses cardiac neovascularization by down-regulating VEGF and PlGF expression, Cardiovascular Research, 2014,104(3): 412-422.（IF: 5.94）
(3) Wang N,Wu Y,Zeng N,Wang H,Deng P,Xu Y,Feng Y,Zeng H,Yang H,Hou K, Wang A, Parthasarathy K, Goyal S, Qin G, Wu M*. E2F1 Hinders Skin Wound Healing by Repressing Vascular Endothelial Growth Factor (VEGF) Expression, Neovascularization, and Macrophage Recruitment. PLoS One. 2016 Aug 4. 11(8): e0160411. (IF: 3.057)
(4) Ning Zeng, Haiping Wang, Yi Xu, Yiping Wu and Min Wu*. Effect of histone deacetylase inhibitor, trichostatin A, on cartilage regeneration from free perichondrial grafts in rabbits. Tropical Journal of Pharmaceutical Research, 2017 June, 1596-5996 (print); 1596-9827(electronic). (IF: 0.569)
(5) Ning Zeng, Yi Xu, Yiping Wu, Tang Hongbo and Min Wu. Bryostatin 1 causes attenuation of TPA‐mediated tumor promotion in mouse skin. MOLECULAR MEDICINE REPORTS 17: 1077-1082, 2018. (IF: 1.922)
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